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Transcript segment

Baric Transcribed Interview (Redacted), pp.78-79 · baric_ti:utt:00701

Page text: p.78, p.79 · original PDF

Date
2026-04-10 09:00 (day precision)
Type
transcript segment · interview
recipient
Christina Salazar, Harry Kazenoff, David T. Lambeth III, William Henderson, Clark Ervin, Jake Greenberg
speaker
Ralph S. Baric
Topics
Natural origin / zoonotic spilloverFurin cleavage site and molecular features
Yes. The main problem that the evolutionary biologists were running into was about that time they only had 8 to 15 full-length genomes of the virus DC.Scheduling@LexitasLegal.com sequenced, and they had no other sequences of related viruses. There was a rumor that a pangolin virus existed with 100 percent identity, or close to 100 percent identity to SARS-2. That turned out not to be true, as the pangolin, called the pangolin GD strain, which was about 90 percent identical, I believe, is the virus they were talking about. It has a receptor binding domain that is almost identical, nearly identical to SARS-2, but it does have a couple point mutations in it. And the reason that's important is that that meant that there were natural isolates in nature that had that sequence, which means it's not engineered.

Extracted statements

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StatementGradeAttributionStance
There was a rumor that a pangolin virus existed with 100 percent identity, or close to 100 percent identity to SARS-2. own voice, substantive speaker_own asserts
That turned out not to be true, as the pangolin, called the pangolin GD strain, which was about 90 percent identical, I believe, is the virus they were talking about. own voice, substantive speaker_own asserts
It has a receptor binding domain that is almost identical, nearly identical to SARS-2, but it does have a couple point mutations in it. own voice, substantive speaker_own asserts

In context

A single line often inverts meaning once you see what it answers, so neighbouring messages are always shown.

  1. 2026-04-10 09:00 Harry Kazenoff open
    On the 31st --
  2. 2026-04-10 09:00 Christina Salazar open
    So yeah, I believe there was a call on the 31st, and that was Eddie Holmes, the Aussies reaching out with Kristian Andersen, to let Dr. Fauci know that he thought that, or they were concerned that it was engineered.
  3. 2026-04-10 09:00 Ralph S. Baric open
    You know, being on that call, that was not a decision of that group that it was engineered.
  4. 2026-04-10 09:00 Christina Salazar open
    Did they discuss it?
  5. 2026-04-10 09:00 Ralph S. Baric
    Yes. The main problem that the evolutionary biologists were running into was about that time they only had 8 to 15 full-length genomes of the virus DC.Scheduling@LexitasLegal.com sequenced, and they had no other sequences of related viruses. There was a rumor that a pangolin virus existed with 100 percent identity, or close to 100 percent identity to SARS-2. That turned out not to be true, as the pangolin, called the pangolin GD strain, which was about 90 percent identical, I believe, is the virus they were talking about. It has a receptor binding domain that is almost identical, nearly identical to SARS-2, but it does have a couple point mutations in it. And the reason that's important is that that meant that there were natural isolates in nature that had that sequence, which means it's not engineered.
  6. 2026-04-10 09:00 Christina Salazar open
    Okay.
  7. 2026-04-10 09:00 Ralph S. Baric open
    It was later then found that the BANAL strains on the Cambodian-Thailand-Chinese border --
  8. 2026-04-10 09:00 Christina Salazar open
    This is --
  9. 2026-04-10 09:00 Ralph S. Baric open
    BANAL-52 and BANAL-1-something or another, also had receptor binding domains that were virtually identical to SARS-2. So there's two bad viruses and pangolin virus within the clade, within that branch of SARS-2 that had identical receptor binding domains, which means that it's not engineered.

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