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Initially it came from the Chinese, social media speculation about what was the cause of the pandemic. The question certainly came up, and I can tell you the same thing that I told them, as far as this virus differs from the SARS coronavirus-2 strain by over 6,000 nucleotides, which means that it's incredibly different. So there's no way it could ever be associated with the emergence of this virus. It's just not possible. Same thing with the WIV1 virus. That's been validated by I don't know how many independent investigators around the globe. When they compare the sequence they say it is not the cause of the pandemic.
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Okay. So what else was discussed in the meeting?
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We discussed the P3CO documents. I sort of told them why we felt that the data didn't demonstrate a gain-of-function phenotype, and that was because there was no change or increased replication of the virus in human DC.Scheduling@LexitasLegal.com cells. There was no increased pathogenesis in mice. Alternatively, there was a loss of disease severity in mice, and so that's a loss-of-function phenotype.
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And who made those decisions from NIH?
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2026-04-10 09:00
Ralph S. Baric
Well, again, my job is to send the document to the NIH. It goes into a black box.
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Who did you send it to?
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Oh, that will also be on my email chain. I don't remember her name. But you should have that also in the email documents.
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So you sent all of these documents to NIH after your meeting.
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Yeah. I don't think the university did. I thought it was through me. Do you remember?